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@kaiahfields.bsky.social

11 Followers  |  15 Following  |  2 Posts  |  Joined: 10.03.2025  |  1.5034

Latest posts by kaiahfields.bsky.social on Bluesky

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Masters of adaptation: How cancer and immune cell plasticity mediates tumor progression The ability of cancer cells and immune cells cell to dynamically change their phenotype contributes to tumor progression and therapy resistance. This Essay explores the mutual plasticity of various ce...

Sheera and I co-wrote a review on the mutual plasticity of cancer cells and immune cells! journals.plos.org/plosbiology/...
@heidefordlab.bsky.social

22.07.2025 21:07 β€” πŸ‘ 0    πŸ” 0    πŸ’¬ 0    πŸ“Œ 0

@kaiahfields.bsky.social @heidefordlab.bsky.social

21.05.2025 20:22 β€” πŸ‘ 3    πŸ” 1    πŸ’¬ 0    πŸ“Œ 0
New Publication
EYA3 regulation of NF-ΞΊB and CCL2 suppresses cytotoxic NK cells in the premetastatic niche to promote TNBC metastasis
Connor Hughes, Sheera Rosenbaum, PhD, and Heide Ford, PhD

New Publication EYA3 regulation of NF-ΞΊB and CCL2 suppresses cytotoxic NK cells in the premetastatic niche to promote TNBC metastasis Connor Hughes, Sheera Rosenbaum, PhD, and Heide Ford, PhD

Congratulations to the H. Ford Lab on a new publication, with Connor Hughes and Sheera Rosenbaum, PhD, as co-first authors! Read "EYA3 regulation of NF-ΞΊB and CCL2 suppresses cytotoxic NK cells in the premetastatic niche to promote TNBC metastasis" here: bit.ly/3SNMdgK @heidefordlab.bsky.social

21.05.2025 20:11 β€” πŸ‘ 6    πŸ” 1    πŸ’¬ 0    πŸ“Œ 0
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EYA3 regulation of NF-ΞΊB and CCL2 suppresses cytotoxic NK cells in the premetastatic niche to promote TNBC metastasis EYA3 promotes evasion of cytotoxic NK cells to enable metastasis, highlighting a therapeutic target in metastatic breast cancer.

new publication from the Ford lab!
@heidefordlab.bsky.social
www.science.org/doi/10.1126/...

14.05.2025 16:12 β€” πŸ‘ 3    πŸ” 0    πŸ’¬ 0    πŸ“Œ 0

Sen. Hickenlooper meets with CU Anschutz cancer lab to discuss national medical research cuts buff.ly/2Nps9P7

19.03.2025 01:17 β€” πŸ‘ 3    πŸ” 2    πŸ’¬ 0    πŸ“Œ 0

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