Juliane Da Graça

Endosome–ER contact sites in phagophore formation Autophagy is a crucial ‘self-eating’ mechanism used by eukaryotic cells to degrade and recycle cytosolic materials. A recent study by Da Graça et al. reports that the dynamic mobilization of endosome–...

Endosome–ER contact sites in phagophore formation: Trends in Cell Biology www.cell.com/trends/cell-...

Check out the recent spotlight published in @cp-trendscellbio.bsky.social about our study on ER-endosome contacts in autophagy by @julianedagraca.bsky.social

When you submit papers 6 months appart but they end up being published on the same week 🤯

Amazing collaboration with @carlaalemany.bsky.social to show how Influenza A virus rewires PI4P synthesis 🦠

ER-endosome contacts generate a local environment promoting phagophore formation Da Graça et al. identify ER-endosome contact sites as critical hubs for autophagosome biogenesis, where starvation triggers sequential endosome tethering to ER exit sites. This spatiotemporal coordina...

FINALLY OUT ! After a long journey (of PhD and publication process) my paper on the role of ER-endosomes contact sites in autophagosome formation is officially out 🥳🥳 www.cell.com/cell-reports...

Thank you so much for this PhD prize !! So grateful for this award and for having an amazing autophagic family ! ✨✨

Back to celebrate accepted papers 🥳

The scientific highlights of my first PhD paper and my PhD journey at @inem-necker.bsky.social ☀️

Super communication project made by @biospc.bsky.social students !

🏆 Congrats to @julianedagraca.bsky.social! Former PhD student at INEM, Juliane has been awarded the 2025 Thesis Prize of the @clubexo-endo.bsky.social for her work on ER-endosome contact sites & autophagy regulation.
👏 Well done, Juliane! @membramics-lab.bsky.social

Super happy to be awarded the Thesis Prize 2025 from @clubexo-endo.bsky.social 🤩

First time making a cover !! So proud 🤩

Check this review if you want to know more about endolysosomal morphodynamics upon stress!

New BioRxiv manuscript on IAV infection and phosphoinositides turnover !

Influenza A virus-induced production of PI4P at the endoplasmic reticulum involves ATG16L1 and promotes the egress of viral ribonucleoproteins

✍️ @carlaalemany.bsky.social @julianedagraca.bsky.social @membramics-lab.bsky.social JB Brault, N Naffakh & coll #INEM @pasteur.fr

shorturl.at/iLVaf